PND-1186
95%
- Product Code: 102086
CAS:
1061353-68-1
| Molecular Weight: | 501.5 g./mol | Molecular Formula: | C₂₅H₂₆F₃N₅O₃ |
|---|---|---|---|
| EC Number: | MDL Number: | ||
| Melting Point: | Boiling Point: | ||
| Density: | Storage Condition: | -20°C |
Product Description:
PND-1186 is primarily recognized for its role in the field of cancer research. It functions as a potent inhibitor of focal adhesion kinase (FAK), a protein that plays a critical role in cell adhesion, migration, and survival. By inhibiting FAK, PND-1186 disrupts signaling pathways that are often hijacked by cancer cells to promote tumor growth, metastasis, and resistance to therapy. This makes it a valuable tool in preclinical studies aimed at understanding the mechanisms of cancer progression and in the development of targeted anticancer therapies. Researchers utilize PND-1186 to explore its potential in reducing tumor invasiveness and enhancing the efficacy of existing treatments, particularly in cancers where FAK overexpression is implicated.
Product Specification:
| Test | Specification |
|---|---|
| Appearance | White To Off-White Solid |
| Purity (%) | 94.5-100 |
| Nmr | Consistent With Structure |
| Infrared Spectrum | Conforms To Structure |
Sizes / Availability / Pricing:
| Size (g) | Availability | Price | Quantity |
|---|---|---|---|
| 0.005 | 10-20 days | $249.17 |
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| 0.025 | 10-20 days | $676.92 |
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| 0.100 | 10-20 days | $2,192.73 |
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PND-1186
PND-1186 is primarily recognized for its role in the field of cancer research. It functions as a potent inhibitor of focal adhesion kinase (FAK), a protein that plays a critical role in cell adhesion, migration, and survival. By inhibiting FAK, PND-1186 disrupts signaling pathways that are often hijacked by cancer cells to promote tumor growth, metastasis, and resistance to therapy. This makes it a valuable tool in preclinical studies aimed at understanding the mechanisms of cancer progression and in the development of targeted anticancer therapies. Researchers utilize PND-1186 to explore its potential in reducing tumor invasiveness and enhancing the efficacy of existing treatments, particularly in cancers where FAK overexpression is implicated.
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