IRAK inhibitor 2
≥97%
- Product Code: 101036
CAS:
928333-30-6
Molecular Weight: | 306.32 g./mol | Molecular Formula: | C₁₇H₁₄N₄O₂ |
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Density: | Storage Condition: | 2-8℃ |
Product Description:
IRAK inhibitor 2 is primarily utilized in research settings to study inflammatory and immune-related pathways. It targets the interleukin-1 receptor-associated kinase (IRAK) family, which plays a critical role in signaling pathways associated with innate immunity. By inhibiting IRAK, this compound helps researchers understand the mechanisms underlying inflammatory diseases such as rheumatoid arthritis, lupus, and sepsis. It is also used to explore potential therapeutic strategies for cancer, as IRAK signaling can contribute to tumor growth and survival. Additionally, IRAK inhibitor 2 is valuable in investigating the role of IRAK in autoimmune disorders and its connection to Toll-like receptor (TLR) signaling. Its application extends to drug discovery efforts aimed at developing targeted therapies for conditions driven by dysregulated immune responses.
Sizes / Availability / Pricing:
Size (g) | Availability | Price | Quantity |
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0.005 | 10-20 days | ฿23,094.00 |
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0.010 | 10-20 days | ฿31,482.00 |
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IRAK inhibitor 2
IRAK inhibitor 2 is primarily utilized in research settings to study inflammatory and immune-related pathways. It targets the interleukin-1 receptor-associated kinase (IRAK) family, which plays a critical role in signaling pathways associated with innate immunity. By inhibiting IRAK, this compound helps researchers understand the mechanisms underlying inflammatory diseases such as rheumatoid arthritis, lupus, and sepsis. It is also used to explore potential therapeutic strategies for cancer, as IRAK signaling can contribute to tumor growth and survival. Additionally, IRAK inhibitor 2 is valuable in investigating the role of IRAK in autoimmune disorders and its connection to Toll-like receptor (TLR) signaling. Its application extends to drug discovery efforts aimed at developing targeted therapies for conditions driven by dysregulated immune responses.
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