JNK-IN-8
98%
- Product Code: 124754
CAS:
1410880-22-6
| Molecular Weight: | 507.59 g./mol | Molecular Formula: | C₂₉H₂₉N₇O₂ |
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| Density: | 1.283±0.06 g/cm3(Predicted) | Storage Condition: | 2-8°C |
Product Description:
JNK-IN-8 is primarily used in research settings to study the c-Jun N-terminal kinase (JNK) signaling pathway, which plays a critical role in cellular stress responses, apoptosis, and inflammation. It is a selective inhibitor of JNK, making it a valuable tool for investigating the biological functions of this kinase in various disease models, including neurodegenerative disorders, cancer, and inflammatory conditions. Researchers utilize JNK-IN-8 to explore potential therapeutic strategies by modulating JNK activity, as its inhibition has been linked to protective effects in conditions like Alzheimer's disease, Parkinson's disease, and ischemia-reperfusion injury. Additionally, it aids in understanding the molecular mechanisms underlying stress-induced cell death and survival pathways. Its application extends to preclinical studies aimed at developing targeted therapies for diseases where JNK signaling is dysregulated.
Sizes / Availability / Pricing:
| Size (g) | Availability | Price | Quantity |
|---|---|---|---|
| 0.005 | 10-20 days | $429.43 |
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JNK-IN-8
JNK-IN-8 is primarily used in research settings to study the c-Jun N-terminal kinase (JNK) signaling pathway, which plays a critical role in cellular stress responses, apoptosis, and inflammation. It is a selective inhibitor of JNK, making it a valuable tool for investigating the biological functions of this kinase in various disease models, including neurodegenerative disorders, cancer, and inflammatory conditions. Researchers utilize JNK-IN-8 to explore potential therapeutic strategies by modulating JNK activity, as its inhibition has been linked to protective effects in conditions like Alzheimer's disease, Parkinson's disease, and ischemia-reperfusion injury. Additionally, it aids in understanding the molecular mechanisms underlying stress-induced cell death and survival pathways. Its application extends to preclinical studies aimed at developing targeted therapies for diseases where JNK signaling is dysregulated.
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