GNF-7
10mM in DMSO
- Product Code: 190211
CAS:
839706-07-9
Molecular Weight: | 547.53 g./mol | Molecular Formula: | C₂₈H₂₄F₃N₇O₂ |
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Density: | Storage Condition: | -20°C |
Product Description:
GNF-7 is a potent small-molecule inhibitor primarily investigated for its antitumor activity. It functions by targeting key signaling pathways involved in cancer cell proliferation and survival, particularly through inhibition of tyrosine kinases and modulation of the heat shock protein 90 (HSP90) pathway. This dual mechanism makes it effective in inducing apoptosis and suppressing the growth of various cancer cell lines, including those resistant to conventional therapies.
It has shown promising results in preclinical studies against hepatocellular carcinoma, leukemia, and other solid tumors. Due to its ability to disrupt oncogenic protein stability and signaling, GNF-7 is used as a research tool in oncology to explore novel therapeutic strategies and combination treatments. Its application extends to studies on drug resistance mechanisms and the development of more selective kinase inhibitors.
Sizes / Availability / Pricing:
Size | Availability | Price | Quantity |
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1ml | 10-20 days | ฿7,060.00 |
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GNF-7
GNF-7 is a potent small-molecule inhibitor primarily investigated for its antitumor activity. It functions by targeting key signaling pathways involved in cancer cell proliferation and survival, particularly through inhibition of tyrosine kinases and modulation of the heat shock protein 90 (HSP90) pathway. This dual mechanism makes it effective in inducing apoptosis and suppressing the growth of various cancer cell lines, including those resistant to conventional therapies.
It has shown promising results in preclinical studies against hepatocellular carcinoma, leukemia, and other solid tumors. Due to its ability to disrupt oncogenic protein stability and signaling, GNF-7 is used as a research tool in oncology to explore novel therapeutic strategies and combination treatments. Its application extends to studies on drug resistance mechanisms and the development of more selective kinase inhibitors.
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