FPS-ZM1
98%
- Product Code: 60249
CAS:
945714-67-0
| Molecular Weight: | 327.85 g./mol | Molecular Formula: | C₂₀H₂₂ClNO |
|---|---|---|---|
| EC Number: | MDL Number: | MFCD22378757 | |
| Melting Point: | Boiling Point: | ||
| Density: | Storage Condition: | -20℃ |
Product Description:
FPS-ZM1 is primarily utilized in research focused on neurodegenerative diseases, particularly Alzheimer's disease. It functions as a selective inhibitor of the receptor for advanced glycation end products (RAGE), which plays a significant role in inflammation and neuronal damage. By blocking RAGE, FPS-ZM1 helps reduce amyloid-beta accumulation and neuroinflammation, making it a promising candidate for therapeutic studies. Additionally, it is used to explore its potential in mitigating cognitive decline and improving memory functions in preclinical models. Its application extends to investigating the molecular mechanisms underlying RAGE-mediated pathways in various neurological disorders.
Product Specification:
| Test | Specification |
|---|---|
| APPEARANCE | whithe solid |
| PURITY | 97.5-100 |
| Infrared spectrum | Conforms to Structure |
| NMR | Conforms to Structure |
Sizes / Availability / Pricing:
| Size (g) | Availability | Price | Quantity |
|---|---|---|---|
| 0.005 | 10-20 days | ฿2,980.00 |
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| 0.025 | 10-20 days | ฿9,700.00 |
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| 0.050 | 10-20 days | ฿14,600.00 |
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| 0.100 | 10-20 days | ฿22,000.00 |
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| 0.500 | 10-20 days | ฿41,000.00 |
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FPS-ZM1
FPS-ZM1 is primarily utilized in research focused on neurodegenerative diseases, particularly Alzheimer's disease. It functions as a selective inhibitor of the receptor for advanced glycation end products (RAGE), which plays a significant role in inflammation and neuronal damage. By blocking RAGE, FPS-ZM1 helps reduce amyloid-beta accumulation and neuroinflammation, making it a promising candidate for therapeutic studies. Additionally, it is used to explore its potential in mitigating cognitive decline and improving memory functions in preclinical models. Its application extends to investigating the molecular mechanisms underlying RAGE-mediated pathways in various neurological disorders.
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