c-Fms-IN-3
≥99%
- Product Code: 99925
CAS:
885704-21-2
Molecular Weight: | 406.52 g./mol | Molecular Formula: | C₂₃H₃₀N₆O |
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EC Number: | MDL Number: | ||
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Density: | Storage Condition: | 2-8℃ |
Product Description:
c-Fms-IN-3 is primarily used in research settings to study and inhibit the activity of the colony-stimulating factor-1 receptor (CSF-1R), also known as c-Fms. This receptor plays a critical role in the regulation of macrophages and other immune cells. By targeting c-Fms, this compound helps researchers investigate its involvement in diseases such as cancer, inflammatory disorders, and autoimmune conditions. In cancer research, it is particularly valuable for exploring tumor-associated macrophages (TAMs), which contribute to tumor growth and immune evasion. Inhibiting c-Fms with this compound can provide insights into potential therapeutic strategies to modulate the tumor microenvironment and enhance anti-cancer immune responses. Additionally, it aids in understanding the mechanisms of macrophage-dependent inflammation, offering potential pathways for treating chronic inflammatory diseases.
Sizes / Availability / Pricing:
Size (g) | Availability | Price | Quantity |
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0.005 | 10-20 days | ฿26,244.00 |
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0.010 | 10-20 days | ฿36,729.00 |
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c-Fms-IN-3
c-Fms-IN-3 is primarily used in research settings to study and inhibit the activity of the colony-stimulating factor-1 receptor (CSF-1R), also known as c-Fms. This receptor plays a critical role in the regulation of macrophages and other immune cells. By targeting c-Fms, this compound helps researchers investigate its involvement in diseases such as cancer, inflammatory disorders, and autoimmune conditions. In cancer research, it is particularly valuable for exploring tumor-associated macrophages (TAMs), which contribute to tumor growth and immune evasion. Inhibiting c-Fms with this compound can provide insights into potential therapeutic strategies to modulate the tumor microenvironment and enhance anti-cancer immune responses. Additionally, it aids in understanding the mechanisms of macrophage-dependent inflammation, offering potential pathways for treating chronic inflammatory diseases.
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