Remodelin is primarily investigated for its potential in targeting age-related cellular changes. It is studied for its ability to influence nuclear architecture by reducing nuclear blebbing, a phenomenon associated with cellular aging. Researchers are exploring its application in mitigating the effects of Hutchinson-Gilford progeria syndrome, a rare genetic disorder that causes accelerated aging. Additionally, it is being examined for its role in improving cellular function and viability in aged or damaged cells, which could have implications for regenerative medicine and anti-aging therapies. Its mechanism involves modulating specific proteins involved in maintaining nuclear integrity, making it a promising candidate for further research in age-related diseases and cellular rejuvenation.