Cinromide

≥99%

Reagent Code: #100100
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CAS Number 58473-74-8

science Other reagents with same CAS 58473-74-8

blur_circular Chemical Specifications

scatter_plot Molecular Information
Weight 254.12 g/mol
Formula C₁₁H₁₂BrNO
inventory_2 Storage & Handling
Storage 2-8℃

description Product Description

Cinromide is a synthetic compound developed as a potential anticonvulsant agent in the pharmaceutical industry. It was investigated for treating epilepsy and other seizure disorders by stabilizing neuronal activity. Preclinical and early clinical research explored its efficacy in reducing the frequency and severity of seizures, but due to concerns such as hepatotoxicity and limited effectiveness, it was discontinued and is not used in current clinical practice. Its proposed mechanism involves modulating neurotransmitter release or ion channel function in the brain. Further studies would be needed to evaluate any therapeutic potential and safety profile, though development has ceased.

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Test Parameter Specification
Appearance Off-White Powder
Purity (%) 99.0-100
NMR Conforms to Structure

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Size Availability Unit Price Quantity
inventory 100mg
10-20 days ฿4,851.00

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Cinromide
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Cinromide is a synthetic compound developed as a potential anticonvulsant agent in the pharmaceutical industry. It was investigated for treating epilepsy and other seizure disorders by stabilizing neuronal activity. Preclinical and early clinical research explored its efficacy in reducing the frequency and severity of seizures, but due to concerns such as hepatotoxicity and limited effectiveness, it was discontinued and is not used in current clinical practice. Its proposed mechanism involves modulating

Cinromide is a synthetic compound developed as a potential anticonvulsant agent in the pharmaceutical industry. It was investigated for treating epilepsy and other seizure disorders by stabilizing neuronal activity. Preclinical and early clinical research explored its efficacy in reducing the frequency and severity of seizures, but due to concerns such as hepatotoxicity and limited effectiveness, it was discontinued and is not used in current clinical practice. Its proposed mechanism involves modulating neurotransmitter release or ion channel function in the brain. Further studies would be needed to evaluate any therapeutic potential and safety profile, though development has ceased.

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